Ethanol-induced metabolomic differences in mice using HRAM Q-TOF analysis

Alcohol consumption continues to be a major risk factor for disease development and is associated with neuropsychiatric disorders, cardiovascular diseases, cirrhosis of the liver, various cancers and fetal alcohol syndrome. Alcohol-specific metabolic responses and the underlying pathophysiology and effects on the brain are poorly understood.
This study considered the effect of ethanol-induced metabolite changes in brain tissue using a mouse model.